PSKH2
protein serine kinase H2

Predicted to enable protein serine/threonine kinase activity. Predicted to be involved in chromatin remodeling. Predicted to be active in cytoplasm. [provided by Alliance of Genome Resources, Apr 2025]

Developmental clusters: GC2
Biological processes 7 terms
Expression (TPM)
PSKH2 — as a Regulated Gene

TFs regulating PSKH2 0 TFs

Transcription factors with Perturb-seq knockdown data for PSKH2. The Binding column indicates whether any binding evidence exists for this TF–gene pair (ChIP-seq or motif footprint peaks). The Mean coef is the average Perturb-seq regression coefficient across active gRNAs (positive = PSKH2 upregulated upon KD; negative = downregulated). The Outlier column indicates whether this gene is in the top or bottom 5% of all TF knockdown effects.

Data: Effect:
TF Mean coef Binding Outlier TF→Gene link

Elements linked to PSKH2

Open chromatin peaks (ATAC-seq) in the genomic neighbourhood of PSKH2, linked by TSS proximity or chromatin conformation (Multiome / HiCAR). Each element overlaps at least one TF ChIP-seq binding site — the TFs column shows how many distinct TFs bind that element.

Accessibility Element Dist. to TSS Link type TFs
chr8:86,059,380–86,060,226 9.4 kb Proximal (<10kb) 24
chr8:86,063,309–86,064,635 5.0 kb Proximal (<10kb) 180
chr8:86,068,911–86,069,915 at TSS At TSS 240
chr8:86,071,505–86,071,672 1.8 kb Proximal (<10kb) 24
chr8:86,072,815–86,073,392 3.1 kb Proximal (<10kb) 109
chr8:86,073,661–86,074,117 4.0 kb Proximal (<10kb) 24

Genome Browser

Genomic view of the PSKH2 locus showing ATAC-seq accessibility and RNA-seq expression across the ESC → DE time course, together with TF binding peaks and element-to-TSS loop connections.

chr8:86,049,380 – 86,084,117
Proximal 1 kb Distal 10 kb Multiome HiCAR ATAC-seq RNA-seq