ZNF702P
zinc finger protein 702, pseudogene

Predicted to enable DNA-binding transcription factor activity. Predicted to be involved in regulation of DNA-templated transcription. Predicted to be located in nucleus. [provided by Alliance of Genome Resources, Jul 2025]

Developmental clusters: GC7
Biological processes 4 terms
Expression (TPM)
ZNF702P — as a Regulated Gene

TFs regulating ZNF702P 0 TFs

Transcription factors with Perturb-seq knockdown data for ZNF702P. The Binding column indicates whether any binding evidence exists for this TF–gene pair (ChIP-seq or motif footprint peaks). The Mean coef is the average Perturb-seq regression coefficient across active gRNAs (positive = ZNF702P upregulated upon KD; negative = downregulated). The Outlier column indicates whether this gene is in the top or bottom 5% of all TF knockdown effects.

Data: Effect:
TF Mean coef Binding Outlier TF→Gene link

Elements linked to ZNF702P

Open chromatin peaks (ATAC-seq) in the genomic neighbourhood of ZNF702P, linked by TSS proximity or chromatin conformation (Multiome / HiCAR). Each element overlaps at least one TF ChIP-seq binding site — the TFs column shows how many distinct TFs bind that element.

Accessibility Element Dist. to TSS Link type TFs
chr19:52,993,107–52,993,350 180 bp At TSS 58
chr19:52,993,475–52,994,192 at TSS At TSS 160
chr19:52,997,394–52,997,773 3.9 kb Proximal (<10kb) 31

Genome Browser

Genomic view of the ZNF702P locus showing ATAC-seq accessibility and RNA-seq expression across the ESC → DE time course, together with TF binding peaks and element-to-TSS loop connections.

chr19:52,983,107 – 53,007,773
Proximal 1 kb Distal 10 kb Multiome HiCAR ATAC-seq RNA-seq