LXN
latexin

This gene encodes the only known protein inhibitor of zinc-dependent metallocarboxypeptidases. The encoded protein, latexin, downregulates the population size of hematopoietic stem cells. This protein is found to be downregulated in cancer cells because of promoter hypermethylation. [provided by RefSeq, Jul 2020]

Biological processes 5 terms
Expression (TPM)
LXN — as a Regulated Gene

TFs regulating LXN 0 TFs

Transcription factors with Perturb-seq knockdown data for LXN. The Binding column indicates whether any binding evidence exists for this TF–gene pair (ChIP-seq or motif footprint peaks). The Mean coef is the average Perturb-seq regression coefficient across active gRNAs (positive = LXN upregulated upon KD; negative = downregulated). The Outlier column indicates whether this gene is in the top or bottom 5% of all TF knockdown effects.

Data: Effect:
TF Mean coef Binding Outlier TF→Gene link

Elements linked to LXN

Open chromatin peaks (ATAC-seq) in the genomic neighbourhood of LXN, linked by TSS proximity or chromatin conformation (Multiome / HiCAR). Each element overlaps at least one TF ChIP-seq binding site — the TFs column shows how many distinct TFs bind that element.

Accessibility Element Dist. to TSS Link type TFs
chr3:158,570,578–158,571,791 101.5 kb Distal (>10kb) Multiome 632
chr3:158,643,965–158,644,957 28.1 kb Distal (>10kb) Multiome 928
chr3:158,671,766–158,673,071 14 bp At TSS Multiome 479
chr3:158,731,475–158,733,286 59.7 kb Distal (>10kb) Multiome 470
chr3:158,801,057–158,802,803 129.4 kb Distal (>10kb) Multiome 932
chr3:158,905,172–158,906,095 232.8 kb Distal (>10kb) Multiome 165

Genome Browser

Genomic view of the LXN locus showing ATAC-seq accessibility and RNA-seq expression across the ESC → DE time course, together with TF binding peaks and element-to-TSS loop connections.

chr3:158,560,578 – 158,916,095
Proximal 1 kb Distal 10 kb Multiome HiCAR ATAC-seq RNA-seq