ESAM
endothelial cell adhesion molecule | W117m
ESAM — as a Regulated Gene

TFs regulating ESAM 0 TFs

Transcription factors with Perturb-seq knockdown data for ESAM. The Binding column indicates whether any binding evidence exists for this TF–gene pair (ChIP-seq or motif footprint peaks). The Mean coef is the average Perturb-seq regression coefficient across active gRNAs (positive = ESAM upregulated upon KD; negative = downregulated). The Outlier column indicates whether this gene is in the top or bottom 5% of all TF knockdown effects.

Data: Effect:
TF Mean coef Binding Outlier TF→Gene link

Elements linked to ESAM

Open chromatin peaks (ATAC-seq) in the genomic neighbourhood of ESAM, linked by TSS proximity or chromatin conformation (Multiome / HiCAR). Each element overlaps at least one TF ChIP-seq binding site — the TFs column shows how many distinct TFs bind that element.

Accessibility Element Dist. to TSS Link type TFs
chr11:124,751,748–124,752,579 9.7 kb Proximal (<10kb) 211
chr11:124,758,225–124,760,203 2.1 kb Proximal (<10kb) 594
chr11:124,762,016–124,763,488 at TSS At TSS 595

Genome Browser

Genomic view of the ESAM locus showing ATAC-seq accessibility and RNA-seq expression across the ESC → DE time course, together with TF binding peaks and element-to-TSS loop connections.

chr11:124,741,748 – 124,773,488
Proximal 1 kb Distal 10 kb Multiome HiCAR ATAC-seq RNA-seq